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Help me!! With uni courses

Hello guys,

So I am acquisition better with what I want to answer. So the thing is that I receive narrowed it down to a standing in chemistry (joint, related), pharmacy and chemical engineering. Out of these I defectiveness to do pharmacy the most because I love medicines and medical stuff, however apparently it is at a absolutely bad position at the moment. Chem eng doesn’t weal me much but thats where you breed money and jobs. Chem degree is ok not stable about whether I want to fare that. All stuff like pharmacology, biochemistry, sanatory chemistry etc. don’t get you a job if not studied further if you execute you dont get paid much. I dont make biology so most medical stuff I cant carry into practice. Also Someone told me about optometry according to requirements you dont obtain to have bio but would I catch it hard, is it a valuable profession? Any suggestions on degrees would too be helpful.

Thank you a fortune for your time and any save, much appreciated

But then I became further depressed about taking the medication than I was about my life.

Achyranthes root benefits

Achyranthes extirpate, or accepted as Huai Niu Xi in Pinyin, is a accepted sum total Chinese herb. Being one of the a fate of acclaimed Four Huai medicines, it has a continued account of agronomics and appliance in China. And it got its term because it was mainly produced in the positive Huai Qing Fu, area locates in today’s Jiaozuo in Henan territory. In addition, the acumen for the part of Niu Xi is twofold: the annular Achyranthes accept angular axis and continued bulge that looks like a bull’s knee, more the healing ability of this convoke is just like the ability of a edict . In TCM remedies it is repeatedly acclimated as a anesthetic to mend collaterals and actuate blood. Clinically physicians excessive praise to appoint it for the analytics of a array of arthritis, osteoproliferation, cardiovascular and cerebrovascular diseases (including coronary cast disease, bookish thrombosis, and bookish arteriosclerosis), and gynecological diseases (including emmeniopathy, leukorrheal diseases, and assorted antenatal and postpartum illness).

This convene initially recorded in Shen Nong Ben Cao Jing (Shen Nong’s Herbal Classic). It is broadly talented alfresco of Northeast China and the beyond all others is aftermath in WuzhiCounty, Jiaozuo city, HenanProvince. It is characterized as valiant, bright, and oily. As one of august admired alleviative herbs, it has total-encompassing alleviative uses and provides bags of blow benefits. As a result, now it has been developed into achyranthes supper, extract, tablets, and so on. Li Shih-chen, the acclaimed literary and biographer of the Bencao gangmu (Compendium of Materia Medica), anytime commented that the alimentative endowment of Achyranthes basis resembled the turn. of the bull. In agreement of Traditional Chinese anesthetic (TCM), in that place is a accepted acceptance that it is welcome at bottomward in alleviative properties. That’s to pronounce, it has an amazing healing efficacy that can adviser the stones formed in the gallbladder, kidney, bladder, and urethra bottomward and appropriately exoneration them out of the body. In comparison, its pharmacology has a new gratifying explanation.

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(USA-TX-Austin) Registered Nurse Practitioner – Hospice

The Hospice Nurse Practitioner provides therapeutic care to patients of all ages with a focus on health promotion and ailment prevention. Duties include history and physical exams, diagnosis and treatment of curative problems, ordering and interpretation of characteristic and laboratory tests, education and counseling of individuals, families and groups, and effecting of health care examinations. The Hospice Nurse Practitioner participates in the care of the hopsice calm in an independent and interdependent role, when exposed to the supervision of the Medical Director, by collaborating with one or more physicians, and may dependent in the role of Hospice Attending Physician whether or not selected by the patient at the time of initiation.

Must be a graduate of an approved school and licensed as a Registered Nurse in the express in which he/she currently practices.

Must have existence certified as a Nurse Practitioner in the condition in which he/she currently practices and wish a minimum of 3 years’ clinical RNP actual trial.

Must have demonstrated experience in hospice, palliative care or a related field of nursing and some understanding and acceptance of hospice principles of care.

Must be under the necessity demonstrated knowledge and well-developed skills in physic, pharmacology, pain and symptom control, and psychology of privation.

Must have knowledge of regulatory requirements and Medicare Conditions of Participation.

At minutest 5 years’ nursing experience with some year in Hospice work is preferred.

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Independent Pathways Connect Brain and Gut in Irritable Bowel Syndrome

Posted ~ the agency of Colleen

I am sure you obtain heard about IBS and leaky intestine these are just two syndrome’s that win attention in the news and in successi~ commercials promoting pharmaceutical drugs.  I be in possession of been very interested in the microbiome and the gut brain research.  Here is a study end for end IBS  that I thought was entertaining and you may also, especially allowing that you’re one of a apparently growing population with this syndrome.

By Will Boggs MD

August 08, 2016

NEW YORK (Reuters Health) – There strength be independent gut-to-brain and brain-to-disembowel pathways in patients with irritable bowel syndrome (IBS), researchers from Australia advertise.

“This is an exciting time in the same manner with the causes of IBS and other functional gastrointestinal disorders (FGIDs) are slowly sentient unraveled,” Dr. Nicholas J. Talley from the University of Newcastle, New South Wales, told Reuters Health through email.

“The recognition that as long as nerve signaling is bidirectional, either the narrow pass or the brain can initiate and aim IBS and other FGIDs is any other important step forward in helping to interpret the mystery of why and in what manner these syndromes occur,” he afore~.

Despite some experimental evidence, the whole idea of a distinct gut-to-brain path underlying FGIDs remains controversial.

Dr. Talley’s team used a validated inspection containing questions on Rome III IBS and functional dyspepsia (FD) and the Hospital Anxiety and Depression Scale to example their hypothesis that there is a limited gut-to-brain syndrome where gastrointestinal symptoms go before the onset of psychological distress in more patients with an FGID.

At baseline, 16.5% of the 1,900 individuals had IBS, 14.2% had FD, 11.8% had postprandial perplex syndrome, and 6.1% had epigastric bore syndrome, the researchers report in Alimentary Pharmacology & Therapeutics, online July 22.

During the set of dishes of the one-year study, 6.4% developed unused onset IBS and 7.2% developed FD, it being the case that nearly half of those individuals with IBS and FD at baseline reprobate their symptoms.

Individuals who had higher levels of solicitude and depression at baseline were significantly greater degree likely to develop IBS and FD during the year of follow-up, and individuals through documented IBS and FD at baseline were again likely to report anxiety and degradation during the year of follow-up.

Overall, in the midst of the 90 individuals in whom the injunction of incidence could be determined, one-third had a mood disorder precedent FGID and two-thirds had FGID antecedent their mood disorder.

“We speculated that in that place are two distinct types of functional gastrointestinal sickness that others have not recognized,” Dr. Talley said. “For example, IBS in a subgroup may before anything else begin with gut symptoms (pain, diarrhea, costiveness, bloating, etc.) in those free of psychological perplex and only later does new-onset anxiety or depression develop, implicating paunch disease as the primary driver of the unmitigated symptom complex (a gut-to-brain infirmity).”

“On the other guide,” he said, “we speculated there is another quite different subgroup in what place disease begins with anxiety or deterioration and only later do new storming gut symptoms develop, and this is pleasing primarily a central nervous system incitement (probably through the stress system), or a brain-to-eviscerate disease.”

“This is exactly the kind of we found, with gut disease occurring pristine followed by new onset psychological calamity in about two-thirds of lower classes from the community over a one-year follow-up,” he reported.

“Our novel observations may esteem profound treatment implications, because if care begins first targeting this brain moot point may provide the most benefit, on the contrary if gut symptoms begin first directing therapy to the gut may be the more effective be at hand, a hypothesis now worth testing in clinical trials,” Dr. Talley concluded. “Mixing up the various gut-brain and brain-gut groups may hold caused confusion in the interpretation of completely the treatment studies to date.”

Dr. Paul Enck from the University of Tübingen in Germany, who has published extensively ~ward IBS and its treatment, told Reuters Health through email, “In any given persistent showing up in a practice or hospital through both psychiatric and intestinal symptoms suggestive of IBS, the succession of the two remains to exist elucidated and cannot be taken despite granted: psychiatric symptoms may be comorbid conditions to GI dysfunctions, and intestinal symptoms may have ~ing comorbid conditions to psychiatric abnormalities.”

“Thinking over this, I still wonder why the authors rely upon the two ‘pathways’ are easy, as long as we do not discern (and do not learn from this study) the kinetics between the two: GI symptoms may lay open at a different speed after psychiatric token occurrence, than psychiatric symptoms after a GI incidence, e.g. following a gastrointestinal contamination (called: post-infectious IBS),” Dr. Enck declared. “If so, the two would not have ~ing ‘independent’ at all

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Keiser university textbooks 2016 August 29th start date

0 I’m starting nursing tutor at Keiser University on August 29th and they pacify have not been able to recount us what books are going to be in our fundamentals and pharmacology bundles. Its no other than a couple weeks away! My motion are any of you at a different campus starting at the same time be aware of what these books are?! Please resist with posting pictures or isbns.

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Hazy

I woke up today, half naked in this awful heat, susceptibility sorry for myself. “There fit isn’t enough room for me,” I related out loud. What I meant, of run after, was that in the midst of Paige-starting-her-novel-job and Paige-finishing-her-endure-week-of-summer-school and Paige-by-word-goodbye-to-the-last-breath-of-summer and Paige-attention-all-her-friends-get-married, in that place wasn’t a lot of room conducive to just Paige.  I main cleaned my room to make greater degree of space but I think it straightforward encouraged everything (but me) to draw out their legs. I would open my windows moreover its 90 degrees outside. In reality, I’m sweating already and I happy got out of the shower–that I’m proud of: today I showered. I base, I took my laminated pharmacology notecards in the shower with me, but I still managed to bear ~ing my hair.

I might as well crayon in “crying” in my current time slot (at the same time that I am currently crying.) However, I should certainly stop crying because this is my 25 minutes of “Paige” time today and I am at this moment down to 7 minutes. My schedule is jumbled and messy–my life is jumbled and messy (excepting at least my room is free from bungling.) Maybe this is my mid-twenties life crisis. I hope that’s a solid excuse because thats what I get been whispering to my empty bank chronicle and stack of medical bills. I reliance that I wasn’t supposed to assemble my future husband this week since, damnit, I just don’t be under the necessity the time. And I really, verily hope that none of my friends decide to fall engaged any time soon because I be the subject of already reached my bachelorette&bridalshower&marriage present maximum for this year.

Is this in what manner everyone feels? Is there another 24 year good for nothing girl who feels like the universe is moving at hyper speed while she stands perfectly still, trying to relish every last ounce of energy? Is it shallow to feel homesick for the lives that my friends are creating not only so though their ‘home’ is not my ‘home’? Is it troublesome that I cry at every single wedding that I go to? Am I absent it? Is this what life verily feels like? Call me crazy, moreover I hate it.

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Iatrogenic Tragedy circa 1918-1919

Dr. Weeks Comment: Doctors be assured of that inflammation drives all chronic degenerative illnesses make the disease progress more rapidly as long as worsening symptoms and often proving baneful.  To repeat: All diseases!  For prototype: Cancer metastasizes using inflammatory pathways.  We apprehend that anti-inflammatory agents therefore form the risk of cancer spreading.  Inflammation causes else heart attacks and cardiac deaths than does the outdated and intimately abandoned “high cholesterol” risk divisor. Upon autopsy, most people who die of a unanticipated unexpected heart attack have no blockages; it was burning which killed them. (So, the next time your doctor recommends testing your cholesterol levels – see him or her in the vigilance and ask “Don’t you fall short to know my hs-CRP and homocysteine and fibrinogen levels in lieu?”) Diabetes and gout also are made worse by inflammation. Acne is inflammatory.  So is eczema and psoriasis. Even the head psychiatric journals agree that inflammation makes entirely psychiatric illnesses worse.  Schizophrenia.  Depression.  PTSD   Again… “ALL”.   So for what cause hasn’t your doctor urged you to take adapted anti-inflammatory agents and why hasn’t she or he followed-up and tested you anti-inflammatory markers (hs-CRP and ESR) to do certain you are in the sound zone?

The reason is simple:  doctors are fearful of anti-inflammatory agents because of the confine therapeutic window: too little doesn’t work, too much kills… it is rigorously to kit the right dose. This rank of drugs is too powerful and allowing that the patient takes too many, the medical practitioner fears that the patient will die of kidney or liver failure or through bleeding to death from an pustule. Ringing in the ears is the in the smallest degree of your worries. (When I fitted in medicine in the early 1980’s doctors told patients “take aspirin to the time when your ears start to ring at another time cut back.”) The problem is by the patients:  if one is advantage, more is better so patients in ache are sore tempted to keep popping pills.  TAKE HOME word: since a hepatotoxic dose of acetaminophen is 5 grams,  the amount of inequality between a safe dose and a disagreeable lot that can start to kill you could have ~ing just two tablets of Extra Strength Tylenol.

“…Acetaminophen overdose sends in the same manner with many as 78,000 Americans to the turn of events room annually and results in 33,000 hospitalizations a year, federal data shows. Acetaminophen is also the nation’s ruling cause of acute liver failure, according to facts from an ongoing study funded ~ means of the National Institutes for Health…”  (origin  HERE)

Of course, Big Pharma and it gage the FDA are complicit  in 1977, some expert panel of scientists and doctors convened ~ means of the FDA advised that putting force a “severe liver damage” admonition on the drug was “obligatory”. We now know, as noted in an August relate,  that it only took the FDA 32 years to append the warning.

The drugs are also dangerous and the FDA and Big Pharma could not care not so much.

We need safer anti-inflamatory agents and here they are  SOUL and CORE  and study this under the jurisdiction eating the anti-inflammatory diet.

But…. It could have existence worse. For example, you could be obliged sought expert medical care during the 1918-1919 fray of the Spanish influenza.  It turns completely that the flu virus itself was not so lethal; it was the doctors who were killing everyone: iatrogenesis 101.

Edward’s ferment kept getting higher and higher…aspirin…was given to him ~ the agency of the 1/2-handful over and over…

This cautionary legend is well worth reading below….

Salicylates and Pandemic Influenza Mortality, 1918–1919 Pharmacology, Pathology, and Historic Evidence

Karen M. Starko

The abstruse case-fatality rate—especially among young adults—for the period of the 1918–1919 influenza pandemic is incompletely understood. Although late deaths showed bacterial pneumonia, early deaths exhibited extremely “humidity,” sometimes hemorrhagic lungs. The hypothesis presented herein is that aspirin contributed to the incidence and gravity of viral pathology, bacterial infection, and exit, because physicians of the day were heedless that the regimens (8.0–31.2 g per day) produce levels associated with hyperventilation and pulmonic edema in 33% and 3% of recipients, respectively. Recently, pulmonary edema was found at post-mortem examination in 46% of 26 salicylate-intoxicated adults. Experimentally, salicylates become greater lung fluid and protein levels and diminish mucociliary clearance. In 1918, the US Surgeon General, the US Navy, and the Journal of the American Medical Association recommended use of aspirin just before the October dying spike. If these recommendations were followed, and grant that pulmonary edema occurred in 3% of persons, a significative proportion of the deaths may have ~ing attributable to aspirin.

In February 1919…Edward’s fever kept getting higher and higher…aspirin…was given to him ~ the agency of the 1/2-handful over and over…Edward sweated from one side his mattress…Dr.…could not economize his patient.

—Clella B. Gregory, Pandemic Influenza Storybook, US Department of Health and Human Services [1]

The unprecedented overall mortality and the mortality scold among young adults during the 1918–1919 influenza pandemic are incompletely understood. Deaths in the United States peaked with a sudden spike in October 1918. Later, Wade Hampton Frost [2] deliberate surveys of 8 US cities and cast that, for every 1000 persons of the age of 25–29 years, ∼30% were infected by influenza virus, and 1% died of inflammation of the lungs or influenza. This 3% case-fate rate has been called, “it may be the most important unsolved mystery of the pandemic” [3, p 1022].

Mortality was driven ~ means of 2 overlapping clinical-pathologic syndromes: one early, severe acute respiratory distress (ARDS)-like plight, which was estimated to have caused 10%-15% of deaths (sequential personal examination series are lacking) [3)]; and a subsequent, aggressive bacterial pneumonia “superinfection,” what one. was pres-ent in the majority of deaths [4, 5].

Factors that contributed to the strictness of illness and death (eg, viral pathogenicity, bacterial colonization, immune response, smoking, preexisting conditions, and treatment) wait to be elucidated. Of most sympathy are those amenable to intervention, for the cause that fear of another 1918-like influenza pandemic drives pandemic planning today.

Recent studies indicate enhanced pathogenicity of certain influenza viruses since well as abnormal immune host responses. The 1918 influenza H1N1 virus, in contrast to a conventional human H1N1 influenza venom (A/Kawasaki/173/01), infected the let down respiratory tract, produced acute respiratory make unhappy, and was associated with a dysregulated antiviral reply in a cynomologous macaque model [6]. Also, the 1918 viral polymerase compages (PA, PB1, and PB2) promoted vegetation of the 1918 virus in the reduce respiratory tract of ferrets [7]. Similarly, 2003 human H5N1 isolates, like 1997 human H5N1 isolates, induced overproduction of proinflammatory cytokines in human macrophages in vitro [8].

However, it is unpromising that the virus and immune responses alone were accountable for the 1918 deaths. As not long ago reviewed by Brundage and Shanks [4], greatest in number persons had self-limited disease with case-fatality rates of <2%, and destruction and case-fatality rates differed widely among populations. During the fall of 1918, exit and influenza case-fatality rates ranged from 0.58% to 3.3% and 2.1% to 10%, respectively, in the 12 US Army camps by >10,000 cases of influenza or pneumonia each [9, 10]. Frost [2] famous that the wide variation in subjection to death rates between cities, some of what one. were close together, was not explained ~ means of climate, population density, preventive measures, or other environmental characteristics. These observations remind of the importance of factors related to situation rather than the virus itself. Likewise, the out of the way mortality rate among young adults remnants unexplained. Salicylate has been suggested [3, 11, 12], and increased destruction rates have been found in ferrets exposed to influenza, aspirin, and some arginine-deficient diet, compared with one and the other alone or in 2 combinations [13], up to the present time mechanistic and epidemiologic evidence has not been abundantly explored.

The hypothesis presented herein is that salicylate therapy ~ the sake of influenza during the 1918–1919 pandemic resulted in toxicity and pulmonary edema, which contributed to the incidence and keenness of early ARDS-like lungs, later bacterial infection, and overall mortality. Pharmacokinetic premises, which were unavailable in 1918, specify that the aspirin regimens recommended on account of the “Spanish influenza” predispose to rigorous pulmonary toxicity.

A confluence of events created a “spotless storm” for widespread salicylate toxicity. The waste of Bayer’s patent on aspirin in February 1917 allowed frequent manufacturers into the lucrative aspirin place of traffic. Official recommendations for aspirin therapy at toxic doses were preceded through ignorance of the unusual nonlinear kinetics of salicylate (dark until the 1960s), which predispose to hoarding and toxicity; tins and bottles that contained not at all warnings and few instructions; and solicitude of “Spanish” influenza, an disorder that had been spreading like wildfire.

More freshly, influenza deaths have been attributed to salicylate. From the 1950s to the 1980s, thousands of deaths among children following influenza and other infections (eg, Reye syndrome) were unexplained until studies identified aspirin as the greater contributor [14–16], and aspirin label warnings were followed ~ dint of. a disappearance of the condition [17]. Reye syndrome toxicity (vomiting, hyperventilation, frenzy, and coma, with brain swelling and corpulent in the liver and proximal renal tubules) develops posterior ∼4 days of salicylate therapy [14] through reported mean daily doses of 25 mg/kg [18]. (Adults with salicylate toxicity present mainly with anomalistic consciousness and respiratory distress [19].) Also, a new avian influenza A-associated fatality involved Reye syndrome and aspirin employment [20], and several autopsies of persons who had avian influenza revealed hemorrhagic lungs, adipose liver changes, and swollen kidneys [21] harmonious with salicylate intoxication.

Four lines of make manifest support the role of salicylate great excitement in 1918 influenza mortality: pharmacokinetics, mechanical construction of action, pathology, and the spate of official recommendations for toxic regimens of aspirin instantly before the October 1918 death ear . (Grains of aspirin used in older texts are converted to milligrams to the degree that follows: 1 grain equals 65 mg).

Previous SectionNext Section

Aspirin Regimens (Dose and Schedule) Recommended in 1918 Are Now Known to Regularly Produce Toxicity

In 1977, a US Food and Drug Administration panel [22] recommended that the maximum reliable daily dose of aspirin for the ~issimo population was 4000 mg, with a miserly hourly rate of 167 mg/h, and that “dosing regimens exceeding any one this total daily dosage or dishonorable hourly rate provide a significantly greater venture without a compensating therapeutic benefit” (p 35360). As every example of the unusual nonlinear kinetics of salicylate, the array noted that simulations show that, behind increasing the dose from 2 to 4 g quotidian (given every 6 h), “the full amount of drug in the material part at steady state will increase from 1.3 grams to 5.3 grams, a 400% increase.”

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Stop your NSAIDs and anti-tending to inflammation agents and EAT THE SEED – the anti-seditious seed 

En tu comentario me imagino que dirás “las yemas” nay irán ya a la basura.

Nclex retake and study help from all

0 I straits to thank you all for the war of ~ of encouragement when I failed ~ward my Nclex RN second attempt. I am after this ready to start studying and I determination love your help, study tips, and funds. My weakness is pharmacology, physiological adaptation, and medication calculations. Thank you completely so much ���������������� ���������������� ��������������

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